Thursday, July 24, 2008

Etiology of the Avian Bird Flu

According to Dr. Boyd Graves, who has been marginalized over the years by “official” government medical communities for speaking up about the possibility that AIDS was purposefully designed to be an ethnic specific virulent, genocidal agent, Avian flu pandemic could be science run amuck.

Amidst the current Centers for Disease Control media orchestrated public panic over the Avian Flu Pandemic, more and more scientists are beginning to look at the appearance of these lethal virulents as being adapted by science, genetically recombined as biowarfare agents, made in America,” says Graves.

The historical trail traces itself back to the 1843 discovery of the infectious agent (MYCOPLASMA) for the “SPOTTED LEAF DISEASE”, also known as the “TOBACCO MOSAIC VIRUS.” “At the core of human (cancer) disease”, says Graves, is this ‘mycoplasma’ entity that has been weaponized over time realizing lethal and incapacitating conditions for humanity”.

The development of the mycoplasma from plant to animal to human is clear, asserts Graves. “By 1898 the United States had realized the mycoplasma in cows, bovine spongiform encephalitis (BSE). By 1902, they had realized it in horses, equine infectious anemia virua (EIAV), in 1904, they realized it in goats, caprine arthritic encephalitis virus (CAEV). By 1910, they had realized it in fowl, Rous Sarcoma Virus (RSV)”, reports Graves after years and years of committed research into the subject.

According to page 14 – 15 of the 1978 progress report of the U.S. Special program, the 1910 RSV is a product of recombinant genetics. Further review will show the 1918 Spanish Flu was a recombinant bird virus. “The same is true of the 2005 Avian flu’, comments Graves.

Is “recombinant” a natural occurrence in Nature or man made or both?

In 1918, the pandemic began not in a port city of this country, but in the Midwest. Tom Rogers, a whistle-blower, working in the field of virology in the 1990s explains that the scientists who are “in the know” look towards the mid west, in specific, Missouri, to read the pulse of bird carrying plagues. “Birds from Asia and Europe converge at this spot due to the convergence of the Mississippi, Ohio and Missouri river. Various viruses recombine organically”, says Rogers. Rogers tells the story about the Missouri birthplace of the USA born deadly Flu of 1918 claiming that it was purposefully misnamed “Spanish Flu” as a distraction. He tells how A.J. Cohen, a veterinarian in Missouri in 1918 warned meat packers and distributors that a super virus was killing off pullets and piglets. However, Cohen was not only disregarded but his life was threatened by industry. “These meats were sent throughout the nation and ultimately overseas”, claims Rogers.

American soldiers were exposed first to the recombinant and their war travels spread the virus to wherever they went. In fact, more soldiers died from the Spanish Flu than those who died in World War I. Rogers claim is that there is no such thing as “jumping species” as might have been designed and refined by biotechnology techniques as claimed by Dr. Boyd Graves and Dr. Leonard Horowitz, a popular lecturer on the subject and author of AID, EBOLA and other viruses. Rogers claims that the virus passes amongst species due to “wrapping your lips” around it, as he likes to phrase it. As a result of his research into the etiology of non specific species transferred plagues Rogers is now a deeply committed Vegan…that is no meat, no milk. He says the only way you can get the Avian Flu is by “eating” the infected species, or via fluid exchange, like blood transfers, sex, and close saliva contacts, like in AIDS.

Although one can usually find an originating template for bacteria, viruses and drugs in nature, Dr. Boyd Graves claims that US government purposefully brought these lethal viruses to a dangerous new level through biotechnology and laboratory recombinant technologies

When the United States began the Tuskegee syphilis experiments in 1932, they also began the testing of the infectious agent of HIV, the sheep virus VISNA. HIV contains sequences from a 1932 strain of VISNA, strain ks1514. However, in the 1971 progress report (#8) of the U.S. Special Virus (1971), the United States concedes that VISNA had not yet been associated with human disease.

The United States cannot explain how VISNA is the “etiological agent” of HIV, as they report in 1985, 1986 and 1995. See, Science Vol 227, pp. 173 – 7, January, 1985, see also, PROC NAS Vol 83, pp.4007 – 11, June 1986 and PROC NAS Vol 92, pp. 3283 – 87, April 11, 1995.

I believe we can have a much better understanding of medicine and science if we review the U.S. Laboratory of Hygiene (circa 1878), the 1902 Cold Spring Harbor Laboratory, the 1904 Station for Experimental Evolution and the 1926 MIT 'Virus Cancer' Conference”, says Graves. In addition, the May 1946 Appropriations Hearing addresses a "synthetic biological agent" in "useable shape". “This is the recognition of the VISNA agent, which now wreaks havoc across the biomedical world”, comments Graves. Dr. Boyd Graves is certain, that if we review and investigate the U.S. Special Virus program, we will solve not only HIV and AIDS, but we will answer many of the unknown origins of human disease. www.boydgraves.com

Meanwhile, Eileen Dannemann, director of the National Coalition of Organized Women spearheading the collection of the various data wonders if the US government has been recently testing their virus collection on the greyhound dogs…hence the new canine flu epidemic causing CDC concerns about it “jumping species” and infecting humans. Tom Rogers would say don’t kiss your dog. Dr. Boyd Graves and Dr. Len Horowitz would say…just another covert clinical trial.

7 comments:

Dipl.-Ing. Wilfried Soddemann said...

CUT THE CHAIN OF INFECTIONS !

Spread of avian flu by drinking water:

Proved awareness to ecology and transmission is necessary to understand the spread of avian flu. For this it is insufficient exclusive to test samples from wild birds, poultry and humans for avian flu viruses. Samples from the known abiotic vehicles as water also have to be analysed. Proving viruses in water is difficult because of dilution. If you find no viruses you can not be sure that there are not any. On the other hand in water viruses remain viable for a long time. Water has to be tested for influenza viruses by cell culture and in particular by the more sensitive molecular biology method PCR.

Transmission of avian flu by direct contact to infected poultry is an unproved assumption from the WHO. There is no evidence that influenza primarily is transmitted by saliva droplets: “Transmission of influenza A in human beings” http://www.thelancet.com/journals/laninf/article/PIIS1473309907700294/abstract?iseop=true.

There are clear links between the cold, rainy seasons as well as floods and the spread of influenza. There are clear links between avian flu and water, e.g. in Egypt to the Nile delta or in Indonesia to residential districts of less prosperous humans with backyard flocks of birds and without a central water supply as in Vietnam: http://www.cdc.gov/ncidod/EID/vol12no12/06-0829.htm. See also the WHO web side: http://www.who.int/water_sanitation_health/emerging/h5n1background.pdf. That is just why abiotic vehicles as water have to be analysed. The direct biotic transmission from birds, poultry or humans to humans can not depend on the cold, rainy seasons or floods. Water is a very efficient abiotic vehicle for the spread of viruses - in particular of fecal as well as by mouth, nose and eyes excreted viruses. Infected humans, mammals, birds and poultry can contaminate drinking water everywhere. All humans have very intensive contact to drinking water. Spread of avian flu by drinking water can explain small clusters in households too.

Avian flu infections may increase in consequence to increase of virus circulation. Human to human and contact transmission of influenza occur - but are overvalued immense. In the course of influenza epidemics in Germany, recognized clusters are rare, accounting for just 9 percent of cases e.g. in the 2005 season. In temperate climates the lethal H5N1 virus will be transferred to humans via cold drinking water, as with the birds in February and March 2006, strong seasonal at the time when (drinking) water has its temperature minimum.

The performance to eliminate viruses from the drinking water processing plants regularly does not meet the requirements of the WHO and the USA/USEPA. Conventional disinfection procedures are poor, because microorganisms in the water are not in suspension, but embedded in particles. Even ground water used for drinking water is not free from viruses.

In temperate regions influenza epidemics recur with marked seasonality around the end of winter, in the northern as well as in the southern hemisphere. Although seasonality is one of the most familiar features of influenza, it is also one of the least understood. Indoor crowding during cold weather, seasonal fluctuations in host immune responses, and environmental factors, including relative humidity, temperature, and UV radiation have all been suggested to account for this phenomenon, but none of these hypotheses has been tested directly. Influenza causes significant morbidity in tropical regions; however, in contrast to the situation in temperate zones, influenza in the tropics is not strongly associated with a certain season.

In the tropics, flood-related influenza is typical after extreme weather. The virulence of influenza viruses depends on temperature and time. Especially in cases of local water supplies with “young” and fresh influenza-contaminated water from low local wells, cisterns, tanks, rain barrels, ponds, rivers or rice paddies, this pathway can explain H5N1 infections. At 24°C, for example, in the tropics the virulence of influenza viruses in water exists for 2 days. In temperate climates with “older” water from central water supplies, the temperature of the water is decisive for the virulence of viruses. At 7°C the virulence of influenza viruses in water extends to 14 days.

Ducks and rice (paddies = flooded by water) are major factors in outbreaks of avian flu, claims a UN agency: Ducks and rice fields may be a critical factor in spreading H5N1. Ducks, rice (fields, paddies = flooded by water; farmers at work drink the water from rice paddies) and people – not chickens – have emerged as the most significant factors in the spread of avian influenza in Thailand and Vietnam, according to a study carried out by a group of experts from the United Nations Food and Agriculture Organization (FAO) and associated research centres. See http://www.un.org/apps/news/story.asp?NewsID=26096&Cr=&Cr1

The study “Mapping H5N1 highly pathogenic avian influenza risk in Southeast Asia: ducks, rice and people” also concludes that these factors are probably behind persistent outbreaks in other countries such as Cambodia and Laos. This study examined a series of waves of H5N1, a highly pathogenic avian influenza, in Thailand and Vietnam between early 2004 and late 2005. Through the use of satellite mapping, researchers looked at several different factors, including the numbers of ducks, geese and chickens, human population size, rice cultivation and geography, and found a strong link between duck grazing patterns and rice cropping intensity.

In Thailand, for example, the proportion of young ducks in flocks was found to peak in September-October; these rapidly growing young ducks can therefore benefit from the peak of the rice harvest in November-December, at the beginning of the cold: Thailand, Vietnam, Cambodia, Laos – as opposed to Indonesia – are located in the northern hemisphere.

These peaks in the congregation of ducks indicate periods in which there is an increase in the chances for virus release and exposure, and rice paddies often become a temporary habitat for wild bird species. In addition, with virus persistence becoming increasingly confined to areas with intensive rice-duck agriculture in eastern and south-eastern Asia, the evolution of the H5N1 virus may become easier to predict.

Dipl.-Ing. Wilfried Soddemann - Epidemiologist - Free Science Journalist soddemann-aachen@t-online.de http://www.dugi-ev.de/information.html

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